"During the first few months of the coronavirus pandemic, the United States became a nation of novice hermits and amateur epidemiologists. The former battened down the hatches; the latter frantically tried to assess just how much danger we were hiding from. Between sourdough seminars and Zoom meetings, Twitter PhD theses were composed and defended seeking to pin down the “infection fatality rate”: the percentage of infected people, including the undiagnosed, who died of covid-19.
In those early innings, good-faith estimates ranged as high as 3 percent and as low as 0.1 percent. As we got more information, however, the plausible estimates narrowed, and is probably in the range of 0.5 to 1.0 percent.
But with more data, something else has become clear: We’re focusing too much on fatality rates and not enough on the people who don’t die but don’t entirely recover, either."
"My response: It’s from the same mechanisms as those that cause death. If the blood-coagulation mechanism is not treated by heparin or nattokinase, fibrosis (widespread tissue scarring) will occur during the illness-and-recovery timeframe. This is entirely preventable. It’s just not being prevented. That’s a clinical decision by the caregivers, and NOT a different (or unknown) pathology from the Covid-19 virus.
From the Washington Post: Data is beginning to show large percentages of disturbing, possibly long-term syndromes in those who survive having COVID-19, even in those who experienced mild symptoms.
My response: Actually, the data has been there for many months." - Stephen Fowkes