Only 30 years ago marine fish were not bred in captivity. Many books at the time used the phrase "Never bred in captivity" about species we take for granted are spawnable today but in all that time one constant remains: some killifish are very very difficult to breed: The Raddaella complex in Aphyosemion, the Diapterons and Episemion to name a few. Eggs a re few and nearly all fungus despite using the soft water they require. The A. cameronense group are purported to be difficult but many people find the no challenge to get fry. But the batesli/kunzi/splendidus complex is another matter. The outliedr is in Germany they are less of a challenge. Is it somethig in the water? It might be. The water in Germany has something in it that is also in the water in Gabon - Selenium, which is not in most places and has tremensoulsy important bioligcal funcstions, no the lest of which is to prevent fungus. Both aniuamlas ans pamts suquester selenium i the sex cells (both eggs and sperm) to give the next genrait the best chance. Tihs is the reason puming sees are good fo ryuou and why "prairie oysters" are eaten. "They're healthy". Reprodictuve filaue in animalsis often remedied by thr addition of selenium. Let's look at a few rasone why as how thes eappy to exotic Aly9seomi. Chemist of selenium Biockje, s celsn virus bactria parasrs water i gabon water in germamy ware ri the us and uk how to add it how to feed it

At this point in in the 21s century but there's no excuse for any viral infection any more. 7000 years ago in India they figured this out. The Chinese got it 5000 years ago and it's why Astragalus is in literally half of all traditional Chinese medicines. Four years ago at Harvard a research scientist named Lipinski set out to explain the mystery if the strange Ebola immunity that's been known (but I would not say well known; it's not in _The Hot Zone_ which is the only book about Ebola) among people of the coastal forest of west Africa. First encountered in 1976 when Ebola was first documented the immunity ranges up to 33% of some populations as measurd by ZEBOV immunoassay of every tenth person in Gabon by the Pasteur Institute. Taylor in 1995 suggested a mechanism which while not wrong was tangential to the issue, and in a nutshell, in the presence of the right amount of selenium Ebola is unable to make that first chemical bond it needs to make, which is a disulfide bond so it should be obvious if you think about it, to be able to connect to the host cell. If that can't happen it dies. This is no theory it's the molecular biology explanation for an immunity we're long known about but were not able to explain until now. Shortly after this came to light the US quietly changed the level of selenium in the RDA guidelines. Hopefully Canada will address this soon. Especially those in and around the Ebola virus. In Canada that's Manitoba.As of this moment I'm pretty sure nobody in the facility has heard of this yet. By all that's holy somebody prove me worng here, please. Now the catch it works on all viruses. Two brazil nuts a day is all you need to raise serum selenium. This would make flu extinct if we had the wherewithal. Refs: http://www.nytimes.com/2014/09/06/health/ebola-immunity.html http://en.ird.fr/the-media-centre/scientific-newssheets/337-possible-natural-immunity-to-ebola http://www.rexresearch.com/ebola/seleniumebola.htm http://www.journalrepository.org/media/journals/BJMMR_12/2014/Dec/Lipinski632014BJMMR14858.pdf